Minimal models for explaining unbounded increase in fitness
On a prior version of my paper on computational complexity as an ultimate constraint, Hemachander Subramanian made a good comment and question: Nice analysis Artem! If we think of the fitness as a function of genes, interactions between two genes, and interactions between three genes and so on, your analysis using epistasis takes into account only the interactions (second order and more). The presence or absence of the genes themselves (first order) can change the…